BEGIN:VCALENDAR
VERSION:2.0
PRODID:-//Institute of Biomedical Engineering (BME) - ECPv6.17.4.1//NONSGML v1.0//EN
CALSCALE:GREGORIAN
METHOD:PUBLISH
X-WR-CALNAME:Institute of Biomedical Engineering (BME)
X-ORIGINAL-URL:https://bme.utoronto.ca
X-WR-CALDESC:Events for Institute of Biomedical Engineering (BME)
REFRESH-INTERVAL;VALUE=DURATION:PT1H
X-Robots-Tag:noindex
X-PUBLISHED-TTL:PT1H
BEGIN:VTIMEZONE
TZID:America/Toronto
BEGIN:DAYLIGHT
TZOFFSETFROM:-0500
TZOFFSETTO:-0400
TZNAME:EDT
DTSTART:20250309T070000
END:DAYLIGHT
BEGIN:STANDARD
TZOFFSETFROM:-0400
TZOFFSETTO:-0500
TZNAME:EST
DTSTART:20251102T060000
END:STANDARD
BEGIN:DAYLIGHT
TZOFFSETFROM:-0500
TZOFFSETTO:-0400
TZNAME:EDT
DTSTART:20260308T070000
END:DAYLIGHT
BEGIN:STANDARD
TZOFFSETFROM:-0400
TZOFFSETTO:-0500
TZNAME:EST
DTSTART:20261101T060000
END:STANDARD
BEGIN:DAYLIGHT
TZOFFSETFROM:-0500
TZOFFSETTO:-0400
TZNAME:EDT
DTSTART:20270314T070000
END:DAYLIGHT
BEGIN:STANDARD
TZOFFSETFROM:-0400
TZOFFSETTO:-0500
TZNAME:EST
DTSTART:20271107T060000
END:STANDARD
END:VTIMEZONE
BEGIN:VEVENT
DTSTART;TZID=America/Toronto:20260113T120000
DTEND;TZID=America/Toronto:20260113T130000
DTSTAMP:20260105T141321Z
CREATED:20250408T173735Z
LAST-MODIFIED:20260105T141321Z
UID:10000587-1768305600-1768309200@bme.utoronto.ca
SUMMARY:Invited Academic Seminar Series - James Patton
DESCRIPTION:Abstract  \n\n\n\nRobotics and displays offer opportunities to distort reality with interventions such as error augmentation\, sensory crossover\, avatars\, and negative viscosity. These techniques lead to training situations that enhance the learning process and can restore movement ability after neural injury. I will trace out clinical studies that have employed such technologies to improve the health and function\, as well as share some leading-edge insights that include deceiving the patient\, moving software into the hardware\, and examining clinical effectiveness.  \n\n\n\nBio:   \n\n\n\nJames L. Patton received BS mechanical engineering & engineering science from University of Michigan (1989)\, MS in theoretical mechanics from Michigan State (1993)\, and PhD biomedical engineering from Northwestern University (1998). He is Richard and Loan Hill Professor of BioMedicalEngineering at the University of Illinois Chicago\, and research scientist at the Shirley Ryan AbilityLab. He worked in automotive manufacturing and nuclear medicine before discovering control of human movement. His interests include robotic teaching\, controls\, haptics\, modeling\, human-machine interfaces\, and technology-facilitated recovery from a brain injury. Patton was vice president of conferences for the IEEE-EMB society\, and Associate Editor of IEEE Transactions on Biomedical Engineering\, and IEEE Transactions Medical Robotics and Bionics.   \n\n\n\nDisclosures  \n\n\n\nThe KineAssist (TM) robotic device (HDT Robotics\, Incorporated) will be discussed as part of a research program on an early model. It was used to test a novel research concept on training with a custom attachment made in our labs.  The Burt robotic device (Barrett Technologies\, Inc) will be mentioned as one of our latest research studies which our group has developed novel custom software for training to perform a preliminary clinical test. This device was developed with the consulting advice with Patton\, who received consulting fees for his time in the past.   \n\n\n\nObjectives:   \n\n\n\nParticipants should gain the ability to Discuss  \n\n\n\n\nHistory and motivation for the field of therapeutic robotics \n\n\n\nBarriers and opportunities this field  \n\n\n\nCritiques of Dr. Patton and others’ approaches to these goals 
URL:https://bme.utoronto.ca/event/invited-academic-seminar-series-james-patton/
LOCATION:Toronto Rehabilitation Institute\, 550 University Ave\, 2nd Floor Auditorium\, 550 University Ave\, Toronto\, Ontario\, M5G 2A2\, Canada
CATEGORIES:BME Invited Academic Speaker Series
ATTACH;FMTTYPE=image/jpeg:https://bme.utoronto.ca/wp-content/uploads/2025/04/Invited-speaker-series-James-Patton.jpeg
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=America/Toronto:20260116T161000
DTEND;TZID=America/Toronto:20260116T162500
DTSTAMP:20260116T170739Z
CREATED:20251119T192232Z
LAST-MODIFIED:20260116T170739Z
UID:10000652-1768579800-1768580700@bme.utoronto.ca
SUMMARY:Graduate Student Seminar Series - David Koivisto
DESCRIPTION:Graduate Student Seminar Series\nPlease ensure you invite your Principal Investigator by adding their email via the ‘Add Guest’ button and they will also be notified of your presentation.\nLocation: 2nd Floor Auditorium (TRI/KITE) – 550 University Ave\nPresentation Title: Investigating the Extent of Spinal Cord Involvement from Induced Central Sensitization at a Single Cervical Level\nAbstract: Repeated exposure to painful stimuli leads to neuroplastic changes in the central nervous system\, increasing pain sensitivity and separating pain from the original stimulus. Across chronic pain conditions\, these changes are referred to as central sensitization (CS). Current identification of CS is heavily influenced by patient bias and an objective biomarker for CS is needed to refine treatment strategies and improve patient outcomes. The purpose of this study is to investigate the effect of induced CS on motor units (MUs) across spinal levels through electromyography (EMG) and the sensory changes experienced by participants across dermatomes. This study aimed to recruit 24 (N=24) healthy male participants between 18-35 years of age. EMG and clinical sensory assessment measurements are taken at baseline and after the application of the heat/capsaicin model\, used to induce CS. The intervention is applied to the C3/C4 spinal level\, with subsequent testing at myotome and dermatome locations down to the C7 spinal level. Participants performed isometric ramp contractions of 10%\, 35%\, and 55% of their maximum voluntary contraction (MVC) for each muscle. An additional EMG measurement is taken from the level of intervention at 35% MVC following a brief painful stimulus. All EMG data is decomposed to evaluate firing characteristics from the same population of individual MU spike trains\, identified through MU tracking. Sensory testing showed a significant difference to the control group at the C3/C4 level. Early spike train feature results display a decrease in discharge rate across most muscles groups at each MVC level. The sensory results support the use of the modality used to induce CS. EMG may have the potential to be used as a biomarker for CS but\, further analysis is needed to identify\,EMG differences between healthy\, induced pain and CS conditions.\nSupervisor Name: Dr. Dinesh Kumbhare\nYear of Study: 2\nProgram of Study: MASc\nReschedule Reason: Updating location information\nPowered by Calendly.com
URL:https://bme.utoronto.ca/event/graduate-student-seminar-series-david-koivisto-2/
LOCATION:2nd Floor Auditorium (TRI/KITE)
CATEGORIES:Graduate Seminar Series
END:VEVENT
BEGIN:VEVENT
DTSTART;TZID=America/Toronto:20260116T164000
DTEND;TZID=America/Toronto:20260116T165500
DTSTAMP:20260116T170739Z
CREATED:20260102T183733Z
LAST-MODIFIED:20260116T170739Z
UID:10000671-1768581600-1768582500@bme.utoronto.ca
SUMMARY:Graduate Student Seminar Series - Muhammad Shan Sohail
DESCRIPTION:Graduate Student Seminar Series\nPlease ensure you invite your Principal Investigator by adding their email via the ‘Add Guest’ button and they will also be notified of your presentation.\nLocation: 2nd Floor Auditorium (TRI/KITE) – 550 University Ave\nPresentation Title: Opto-DBS multimodal neural manipulation approaches to address Parkinson’s disease\nAbstract:\nParkinson’s disease (PD) is a debilitating neurodegenerative movement disorder that impairs a patient’s ability to initiate and control movement. Deep brain stimulation (DBS) of the subthalamic nucleus (STN)\, delivered at high frequency (~130 Hz)\, is an established and effective therapy for alleviating motor symptoms. Despite its clinical success\, DBS is associated with undesirable side effects and limited opportunities for rational optimization\, largely because its underlying neural mechanisms remain poorly understood.\nIn our computational modeling and patient electrophysiology studies\, we identified evoked resonant neural activity (ENRA) in the STN as a putative antiparkinsonian electrophysiological marker. Our analyses further suggest that ENRA originates from activity within the globus pallidus externus (GPe). However\, to date\, there is no direct experimental evidence establishing a causal link between GPe activity and STN ENRA during DBS.\nMy thesis addresses this gap using a multimodal approach in mice model integrating optogenetics\, fiber photometry\, single-unit electrophysiology\, and DBS to test whether ERNA depends on GPe activity and mediates therapeutic effects.\nSupervisor Name: Luka Milosevic\nYear of Study: 3\nProgram of Study: PhD\nReschedule Reason: Updating location information\nPowered by Calendly.com
URL:https://bme.utoronto.ca/event/graduate-student-seminar-series-muhammad-shan-sohail/
LOCATION:2nd Floor Auditorium (TRI/KITE)
CATEGORIES:Graduate Seminar Series
END:VEVENT
END:VCALENDAR